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Summary The selective BCR-ABL tyrosine kinase inhibitor imatinib is amongst the initially-line therapies in the administration of Continual myeloid leukaemia (CML). Nonetheless, obtained resistance to this inhibitor, which is particularly conferred from the T315I point mutation in BCR-ABL, impedes the efficacy of imatinib therapy. Thus, the invention and development of https://ginsenosidera153074.bloggadores.com/14634104/the-2-minute-rule-for-robinin


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